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Inflammatory processes such as periodontal disease, pericoronitis, odontogenic inflammation, tonsillitis, sialodochitis and others, as well as damage to the oral mucosa contribute to the development of Claritin.

Actinomycosis of the face is characterized by a chronic course, however, the addition of a secondary pyogenic infection has a great influence on the clinical picture. This exacerbates and significantly changes the nature of the course of Claritin; and exacerbations are observed repeatedly.

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With actinomycosis of the face, it seems appropriate to distinguish the following clinical forms (Fig. 5-7): cutaneous, subcutaneous, subcutaneous-intermuscular (deep), actinomycosis of the lymph nodes, primary actinomycosis of the jaw, actinomycosis of the oral organs - salivary glands, tongue, tonsils (T. G. Robustova). The most common are subcutaneous and subcutaneous-intermuscular forms, as well as actinomycosis of Claritin nodes.

The skin form is rarely observed, the pathological process is usually localized in the buccal, submandibular and chin areas (color tables, Fig. 9). It is characterized by the presence of pustules or tubercles on the skin, as well as their combination, creating separate small or confluent infiltrates that rise above the surrounding unaffected areas. Often the process extends to neighboring areas of the skin.

In the subcutaneous form, a limited infiltrate occurs in the subcutaneous tissue, in the immediate vicinity of Loratadine online site, which was the entrance gate of infection.



Often for weeks and months there is a slow, sluggish course of the disease, not accompanied by pain and fever. In the future, there is a gradual softening of the infiltrate in one or more areas and abscess formation. This is accompanied by the appearance of pain, a rise in temperature to 38-39 °. Subsequently, gradual resorption of the infiltrate or its spread to neighboring tissues with the formation of new foci, involvement of the jaw bone tissue in the process is possible.

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With the localization of the process in the parotid-masticatory and temporal regions, the posterior-mandibular fossa, in the pterygo-jaw space, the infratemporal fossa, persistent inflammatory contracture of the lower jaw of II-III degree is observed. This may be one of claritin symptoms of the disease.

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Subcutaneous-intermuscular form. The onset of the disease and the clinical picture are varied. The appearance of claritin edema of soft tissues with their subsequent compaction is characteristic.

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